Publication Name	World Journal of Gastroenterology
Manuscript ID	110176
Country	China

Category	Gastroenterology & Hepatology
Manuscript Type	Letter to the Editor
Article Title	Relationship between Helicobacter pylori infection and the development of metabolic dysfunction and associated steatotic liver disease
Manuscript Source	Unsolicited Manuscript
All Author List	Noube Julie Raissa, Shuo-Yi Yao and Fen Wang
Funding Agency and Grant Number	Funding Agency Grant Number National Natural Science Foundation of China 82270594
Corresponding Author	Fen Wang, MD, Professor, Department of Gastroenterology, The Third Xiangya Hospital of Central South University, No. 138 Tongzi Road, Changsha 410013, Hunan Province, China. wfen-judy@csu.edu.cn
Key Words	Helicobacter pylori; Metabolic dysfunction; Metabolic dysfunction-associated steatotic liver disease; Blood glucose; Cholesterol; Body mass index; Blood pressure
Core Tip	This article discussed the relationship between Helicobacter pylori (H. pylori) and the development of metabolic dysfunction and metabolic dysfunction-associated steatotic liver disease (MASLD) based on the study by Ye et al. Their study concluded that H. pylori may play a role in metabolic disturbances (especially in parameters including blood glucose, body mass index, and diastolic blood pressure) and MASLD. There are also other studies in different populations that confirmed the association between H. pylori infection and MASLD.
Citation	Raissa NJ, Yao SY, Wang F. Relationship between Helicobacter pylori infection and the development of metabolic dysfunction and associated steatotic liver disease. World J Gastroenterol 2025; In press

Received	2025-06-03 01:52
Peer-Review Started	2025-06-03 01:52
To Make the First Decision
Return for Revision	2025-06-25 09:45
Revised	2025-07-06 10:31
Second Decision	2025-08-11 02:49
Accepted by Journal Editor-in-Chief
Accepted by Executive Editor-in-Chief	2025-08-13 09:55
Articles in Press	2025-08-13 09:55
Publication Fee Transferred
Edit the Manuscript by Language Editor
Typeset the Manuscript

ISSN	1007-9327 (print) and 2219-2840 (online)
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