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9/29/2014 5:17:00 PM | Browse: 772 | Download: 1125
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Received |
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2014-03-01 11:21 |
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Peer-Review Started |
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2014-03-01 18:36 |
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First Decision by Editorial Office Director |
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2014-04-27 12:24 |
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2014-04-28 00:00 |
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Second Decision by Editor |
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2014-05-26 16:28 |
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Second Decision by Editor-in-Chief |
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Final Decision by Editorial Office Director |
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2014-05-26 16:56 |
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Articles in Press |
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2014-05-26 17:31 |
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Edit the Manuscript by Language Editor |
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2014-05-29 23:55 |
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Typeset the Manuscript |
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2014-09-12 16:41 |
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Publish the Manuscript Online |
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2014-10-01 20:25 |
| Category |
Gastroenterology & Hepatology |
| Manuscript Type |
Editorial |
| Article Title |
Contribution of TLR signaling to the pathogenesis of colitis-associated cancer in inflammatory bowel disease
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| Manuscript Source |
Invited Manuscript |
| All Author List |
Ferenc Sipos, István Fűri, Miklós Constantinovits, Zsolt Tulassay and Györgyi Műzes |
| Funding Agency and Grant Number |
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| Corresponding Author |
Györgyi Műzes, MD, CSc, PhD, 2nd Department of Internal Medicine, Semmelweis University, Szentkirályi Street 46, 1088 Budapest, Hungary. muzes.gyorgyi@med.semmelweis-univ.hu |
| Key Words |
Inflammation; Tissue repair; Immunoregulation; Colitis-associated cancer; Toll-like receptor; Inflammatory bowel disease; Carcinogenesis |
| Core Tip |
Colorectal cancers arising in inflammatory bowel disease patients are considered typical examples of inflammation-associated cancers. The exact role of Toll-like receptor (TLR)-signaling in colitis-associated cancer initiation and development is conflicting. Here we aimed to summarize recent data on the contribution of TLR-mediated immune responses to inflamation-related colonic carcinogenesis. |
| Publish Date |
2014-10-01 20:25 |
| Citation |
Sipos F, Fűri I, Constantinovits M, Tulassay Z, Műzes G. Contribution of TLR signaling to the pathogenesis of colitis-associated cancer in inflammatory bowel disease. World J Gastroenterol 2014; 20(36): 12713-12721
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| URL |
http://www.wjgnet.com/1007-9327/full/v20/i36/12713.htm |
| DOI |
http://dx.doi.org/10.3748/wjg.v20.i36.12713 |
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