Publication Name	World Journal of Gastroenterology
Manuscript ID	10285
Country	China

Received	2014-03-24 20:35
Peer-Review Started	2014-03-24 21:06
To Make the First Decision	2014-04-28 14:15
Return for Revision	2014-05-03 19:31
Revised	2014-05-13 12:53
Second Decision	2014-06-23 10:10
Accepted by Journal Editor-in-Chief
Accepted by Executive Editor-in-Chief	2014-06-23 10:12
Articles in Press	2014-06-23 10:30
Publication Fee Transferred
Edit the Manuscript by Language Editor	2014-08-14 09:14
Typeset the Manuscript	2014-08-29 16:22
Publish the Manuscript Online	2014-09-18 19:50

ISSN	1007-9327 (print) and 2219-2840 (online)
Open Access
Copyright
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Publisher	Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA
Website	http://www.wjgnet.com

Category	Oncology
Manuscript Type	Autobiography
Article Title	Inhibition of KL-6/MUC1 glycosylation limits aggressive progression of pancreatic cancer
Manuscript Source	Unsolicited Manuscript
All Author List	Huan-Li Xu, Xin Zhao, Ke-Ming Zhang, Wei Tang and Norihiro Kokudo
Funding Agency and Grant Number	Funding Agency Grant Number National Natural Science Foundation of China 81302906 Beijing Natural Science Foundation 7144190 New Teacher Fund for Doctor Stations of Ministry of Education of China 20131107120019
Corresponding Author	Ke-Ming Zhang, MD, Department of Hepatobiliary Surgery, 302 Hospital of Chinese People’s Liberation Army, 100, The 4th Ring Road, Beijing 100039, China. zhangkeming302@sina.cn
Key Words	Benzyl-N-acetyl-alpha-galactosaminide; Glycosylation; KL-6/MUC1; Pancreatic carcinoma; Tunicamycin
Core Tip	Aberrant glycosylation of MUC1 is associated with aggressive tumor behavior in many carcinomas. This study evaluated the significance of KL-6/MUC1 glycosylation in pancreatic cancer progression. The results indicate the important involvement of KL-6/MUC1 glycosylation in pancreatic cancer metastasis and invasion, which may be related to the epithelial-mesenchymal transition process. Therapeutic strategies which target glycosylation of KL-6/MUC1 may be useful for controlling the aggressive behavior of pancreatic cancer cells.
Publish Date	2014-09-18 19:50
Citation	Xu HL, Zhao X, Zhang KM, Tang W, Kokudo N. Inhibition of KL-6/MUC1 glycosylation limits aggressive progression of pancreatic cancer. World J Gastroenterol 2014; 20(34): 12171-12181
URL	http://www.wjgnet.com/1007-9327/full/v20/i34/12171.htm
DOI	http://dx.doi.org/10.3748/wjg.v20.i34.12171

Full Article (PDF)	WJG-20-12171.pdf
Full Article (Word)	WJG-20-12171.doc
Manuscript File	10285-Review.doc
Answering Reviewers	10285-Answering reviewers.pdf
Copyright License Agreement	10285-Copyright assignment.pdf
Non-Native Speakers of English Editing Certificate	10285-Language certificate.PDF
Peer-review Report	10285-Peer review.pdf
Scientific Misconduct Check	10285-CorssCheck.jpg
Scientific Editor Work List	10285-Scientific editor work list.pdf