BPG is committed to discovery and dissemination of knowledge
Articles Published Processes
9/18/2014 7:49:00 PM | Browse: 825 | Download: 1161
Publication Name World Journal of Gastroenterology
Manuscript ID 10285
Country/Territory China
Received
2014-03-24 20:35
Peer-Review Started
2014-03-24 21:06
To Make the First Decision
2014-04-28 14:15
Return for Revision
2014-05-03 19:31
Revised
2014-05-13 12:53
Second Decision
2014-06-23 10:10
Accepted by Journal Editor-in-Chief
Accepted by Company Editor-in-Chief
2014-06-23 10:12
Articles in Press
2014-06-23 10:30
Publication Fee Transferred
Edit the Manuscript by Language Editor
2014-08-14 09:14
Typeset the Manuscript
2014-08-29 16:22
Publish the Manuscript Online
2014-09-18 19:50
ISSN 1007-9327 (print) and 2219-2840 (online)
Open Access
Copyright
Article Reprints For details, please visit: http://www.wjgnet.com/bpg/gerinfo/247
Permissions For details, please visit: http://www.wjgnet.com/bpg/gerinfo/207
Publisher Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA
Website http://www.wjgnet.com
Category Oncology
Manuscript Type Autobiography
Article Title Inhibition of KL-6/MUC1 glycosylation limits aggressive progression of pancreatic cancer
Manuscript Source Unsolicited Manuscript
All Author List Huan-Li Xu, Xin Zhao, Ke-Ming Zhang, Wei Tang and Norihiro Kokudo
Funding Agency and Grant Number
Funding Agency Grant Number
National Natural Science Foundation of China 81302906
Beijing Natural Science Foundation 7144190
New Teacher Fund for Doctor Stations of Ministry of Education of China 20131107120019
Corresponding Author Ke-Ming Zhang, MD, Department of Hepatobiliary Surgery, 302 Hospital of Chinese People’s Liberation Army, 100, The 4th Ring Road, Beijing 100039, China. zhangkeming302@sina.cn
Key Words Benzyl-N-acetyl-alpha-galactosaminide; Glycosylation; KL-6/MUC1; Pancreatic carcinoma; Tunicamycin
Core Tip Aberrant glycosylation of MUC1 is associated with aggressive tumor behavior in many carcinomas. This study evaluated the significance of KL-6/MUC1 glycosylation in pancreatic cancer progression. The results indicate the important involvement of KL-6/MUC1 glycosylation in pancreatic cancer metastasis and invasion, which may be related to the epithelial-mesenchymal transition process. Therapeutic strategies which target glycosylation of KL-6/MUC1 may be useful for controlling the aggressive behavior of pancreatic cancer cells.
Publish Date 2014-09-18 19:50
Citation Xu HL, Zhao X, Zhang KM, Tang W, Kokudo N. Inhibition of KL-6/MUC1 glycosylation limits aggressive progression of pancreatic cancer. World J Gastroenterol 2014; 20(34): 12171-12181
URL http://www.wjgnet.com/1007-9327/full/v20/i34/12171.htm
DOI http://dx.doi.org/10.3748/wjg.v20.i34.12171
Full Article (PDF) WJG-20-12171.pdf
Full Article (Word) WJG-20-12171.doc
Manuscript File 10285-Review.doc
Answering Reviewers 10285-Answering reviewers.pdf
Copyright License Agreement 10285-Copyright assignment.pdf
Non-Native Speakers of English Editing Certificate 10285-Language certificate.PDF
Peer-review Report 10285-Peer review.pdf
Scientific Misconduct Check 10285-CorssCheck.jpg
Scientific Editor Work List 10285-Scientific editor work list.pdf