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Articles Published Processes
11/19/2014 3:06:00 PM | Browse: 1307 | Download: 1633
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Received |
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2014-03-27 21:27 |
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Peer-Review Started |
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2014-03-31 09:24 |
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To Make the First Decision |
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2014-05-14 13:42 |
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Return for Revision |
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2014-05-20 14:10 |
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Revised |
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2014-06-03 10:42 |
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Second Decision |
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2014-10-16 15:12 |
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Accepted by Journal Editor-in-Chief |
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Accepted by Executive Editor-in-Chief |
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2014-10-16 15:45 |
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Articles in Press |
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2014-10-16 15:46 |
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Publication Fee Transferred |
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Edit the Manuscript by Language Editor |
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Typeset the Manuscript |
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2014-11-10 13:18 |
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Publish the Manuscript Online |
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2014-11-19 15:06 |
Category |
Immunology |
Manuscript Type |
Review |
Article Title |
Apoptotic signaling through reactive oxygen species in cancer cells
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Manuscript Source |
Invited Manuscript |
All Author List |
Daejin Kim, Ga Bin Park and Dae Young Hur |
Funding Agency and Grant Number |
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Corresponding Author |
Dae Young Hur, MD, PhD, Department of Anatomy and Research Center for Tumor Immunology, Inje University College of Medicine, Bokjiro-75, Jin-gu, Busan 614-735, South Korea. dyhur@inje.ac.kr |
Key Words |
Oxidative stress; Reactive oxygen species; Carcinogenesis; Apoptosis; Signal transduction; Antioxidants |
Core Tip |
Reactive oxygen species originally used to induce injurious cellular effects are now recognized as key physiological molecules for the induction of host defense genes, activation of transcription factors, and regulation of signal transduction. Tumorigenic cells can induce a new redox balance, resulting in cellular adaptation and proliferation. Here, we review the role of oxidative stress in cancer cells using a pathophysiological view. |
Publish Date |
2014-11-19 15:06 |
Citation |
Kim D, Park GB, Hur DY. Apoptotic signaling through reactive oxygen species in cancer cells. World J Immunol 2014; 4(3): 158-173 |
URL |
http://www.wjgnet.com/2219-2824/full/v4/i3/158.htm |
DOI |
http://dx.doi.org/10.5411/wji.v4.i3.158 |
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