Publication Name	World Journal of Diabetes
Manuscript ID	13679
Country	India

Received	2014-08-29 09:49
Peer-Review Started	2014-08-30 13:32
To Make the First Decision	2014-09-28 12:25
Return for Revision	2014-09-30 16:41
Revised	2014-10-14 18:38
Second Decision	2014-12-26 08:39
Accepted by Journal Editor-in-Chief	2014-12-27 00:10
Accepted by Executive Editor-in-Chief	2014-12-31 13:22
Articles in Press	2014-12-31 13:22
Publication Fee Transferred
Edit the Manuscript by Language Editor
Typeset the Manuscript	2015-04-16 12:03
Publish the Manuscript Online	2015-05-15 19:54

ISSN	1948-9358 (online)
Open Access	This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/
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Category	Endocrinology & Metabolism
Manuscript Type	Review
Article Title	Type 2 diabetes mellitus: From a metabolic disorder to an inflammatory condition
Manuscript Source	Invited Manuscript
All Author List	Iqra Hameed, Shariq R Masoodi, Shahnaz A Mir, Mudasar Nabi, Khalid Ghazanfar and Bashir A Ganai
Funding Agency and Grant Number	Funding Agency Grant Number Department of Science and Technology, Government of India to Iqra Hameed Wos-A LS 509/2012
Corresponding Author	Dr. Bashir A Ganai, Professor, Director, Centre for Research and Development, University of Kashmir, Hazratbal Rd, Hazratbal, Srinagar 190006, India. bbcganai@gmail.com
Key Words	Diabetes mellitus; Inflammation; Insulin resistance; β-cell dysfunction; Adipose tissue
Core Tip	Immuno-metabolism, the confluence of meta-bolism and immune system has emerged as a chief breakthrough especially in the field of diabetes mellitus; a metabolic disorder of great magnitude. Activation of immune system by metabolic stress has opened new insights in the pathogenesis and progression of type 2 diabetes (T2D). The link between metabolic overload and activation of the immune system form the core tip of this review. Metabolic stress can cause pathologic activation of the immune system, thus metabolic disorders like T2D manifest and progress as an inflammatory disorder with severe consequences thereof.
Publish Date	2015-05-15 19:54
Citation	Hameed I, Masoodi SR, Mir SA, Nabi M, Ghazanfar K, Ganai BA. Type 2 diabetes mellitus: From a metabolic disorder to an inflammatory condition. World J Diabetes 2015; 6(4): 598-612
URL	http://www.wjgnet.com/1948-9358/full/v6/i4/598.htm
DOI	http://dx.doi.org/10.4239/wjd.v6.i4.598

Full Article (PDF)	WJD-6-598.pdf
Full Article (Word)	WJD-6-598.doc
Manuscript File	13679-Review.docx
Answering Reviewers	13679-Answering reviewers.pdf
Conflict-of-Interest Disclosure Form	13679-Conflict-of-interest statement.pdf
Copyright License Agreement	13679-Copyright assignment.pdf
Peer-review Report	13679-Peer review(s).pdf
Scientific Misconduct Check	13679-CrossCheck.jpg
Scientific Editor Work List	13679-Scientific editor work list.pdf