BPG is committed to discovery and dissemination of knowledge
Articles Published Processes
9/28/2015 11:46:00 AM | Browse: 1228 | Download: 2799
 |
Received |
|
2015-04-09 16:44 |
|
Peer-Review Started |
|
2015-04-09 20:22 |
 |
First Decision by Editorial Office Director |
|
|
|
Return for Revision |
|
2015-05-20 11:08 |
|
Revised |
|
2015-05-26 12:16 |
|
Publication Fee Transferred |
|
|
|
Second Decision by Editor |
|
2015-06-23 18:32 |
|
Second Decision by Editor-in-Chief |
|
|
|
Final Decision by Editorial Office Director |
|
2015-07-03 17:19 |
|
Articles in Press |
|
2015-07-03 17:19 |
|
Edit the Manuscript by Language Editor |
|
2015-08-25 11:41 |
|
Typeset the Manuscript |
|
2015-09-02 11:49 |
|
Publish the Manuscript Online |
|
2015-09-28 11:42 |
| ISSN |
1007-9327 (print) and 2219-2840 (online) |
| Open Access |
This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/
|
| Copyright |
© The Author(s) 2015. Published by Baishideng Publishing Group Inc. All rights reserved.
|
| Article Reprints |
For details, please visit: http://www.wjgnet.com/bpg/gerinfo/247
|
| Permissions |
For details, please visit: http://www.wjgnet.com/bpg/gerinfo/207
|
| Publisher |
Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA |
| Website |
http://www.wjgnet.com |
| Category |
Gastroenterology & Hepatology |
| Manuscript Type |
Basic Study |
| Article Title |
Recql5 protects against lipopolysaccharide/D-galactosamine-induced liver injury in mice
|
| Manuscript Source |
Unsolicited Manuscript |
| All Author List |
Wan-Qin Liao, Ya-Lei Qi, Lin Wang, Xiao-Ming Dong, Tao Xu, Chao-Dong Ding, Rui Liu, Wei-Cheng Liang, Li-Ting Lu, He Li, Wen-Feng Li, Guang-Bin Luo and Xin-Cheng Lu |
| Funding Agency and Grant Number |
| Funding Agency |
Grant Number |
| National Natural Science Foundation of China |
81101472 |
| National Natural Science Foundation of China |
81472556 (to Liao W) |
| National Natural Science Foundation of China |
81372490 (to Lu X) |
| Zhejiang Provincial Natural Science Foundation |
LZ14H160003 (to Lu X) |
| Zhejiang Provincial Program for the Cultivation of High-Level Innovative Health Talents |
to Lu X |
| National Basic Research Program of China (973 Project) |
2011CB504603 |
| Wenzhou Municipal Science and Technology Bureau Foundation |
Y20110090 (to Li H) |
|
| Corresponding Author |
Xin-cheng Lu, Professor, School of Basic Medical Science, Wenzhou Medical University, Chashan Campus, Chashan University Town, Wenzhou 325027, Zhejiang Province, China. xinchenglu@yahoo.com
|
| Key Words |
Recql5; Liver injury; Apoptosis; Oxidative stress; CYP450 |
| Core Tip |
Wild type and Recql5-deficient mice were intraperitoneally injected with lipopolysaccharide and D-galactosamine (LPS/D-Gal). The aim of the study was to explore the effects of Recql5 deficiency on LPS/D-Gal-induced liver injury. Our findings reveal that Recql5 protects against liver injury via inhibition of hepatocyte apoptosis and oxidative stress and regulation of hepatic CYP450 expression levels.
|
| Publish Date |
2015-09-28 11:42 |
| Citation |
Liao WQ, Qi YL, Wang L, Dong XM, Xu T, Ding CD, Liu R, Liang WC, Lu LT, Li H, Li WF, Luo GB, Lu XC. Recql5 protects against lipopolysaccharide/D-galactosamine-induced liver injury in mice. World J Gastroenterol 2015; 21(36): 10375-10384 |
| URL |
http://www.wjgnet.com/1007-9327/full/v21/i36/10375.htm |
| DOI |
http://dx.doi.org/10.3748/wjg.v21.i36.10375 |
All content on this site: Copyright © 1993-2026 Baishideng Publishing Group Inc, its licensors, and contributors. All rights are reserved, including those for text and data mining, AI training, and similar technologies. For all open access content, the relevant licensing terms apply.
