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1/30/2016 5:41:00 PM | Browse: 511 | Download: 764
Publication Name World Journal of Clinical Oncology
Manuscript ID 20142
Country/Territory China
2015-05-29 09:58
Peer-Review Started
2015-05-30 20:42
To Make the First Decision
2015-08-16 17:25
Return for Revision
2015-08-23 19:08
2015-10-16 22:09
Second Decision
2015-12-03 17:34
Accepted by Journal Editor-in-Chief
Accepted by Company Editor-in-Chief
2015-12-11 16:02
Articles in Press
2015-12-11 16:02
Publication Fee Transferred
Edit the Manuscript by Language Editor
2015-12-14 14:44
Typeset the Manuscript
2016-01-18 09:44
Publish the Manuscript Online
2016-01-30 17:42
ISSN 2218-4333 (online)
Open Access This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/
Copyright © The Author(s) 2016. Published by Baishideng Publishing Group Inc. All rights reserved.
Article Reprints For details, please visit: http://www.wjgnet.com/bpg/gerinfo/247
Permissions For details, please visit: http://www.wjgnet.com/bpg/gerinfo/207
Publisher Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA
Website http://www.wjgnet.com
Category Oncology
Manuscript Type Minireviews
Article Title Targeting metabolism in breast cancer: How far we can go?
Manuscript Source Invited Manuscript
All Author List Jing-Pei Long, Xiao-Na Li and Feng Zhang
Funding Agency and Grant Number
Corresponding author Feng Zhang, MD, PhD, Department of Surgery, Hangzhou Women’s Hospital, Kunpeng Road 369, Hangzhou 310008, Zhejiang Province, China. zhang3772@qq.com
Keywords Breast cancer; Targeted therapy; Metabolism; Drug resistance; Chemotherapy
Core Tip Breast cancer cells display distinct metabolic characteristics according to different molecular phenotypes. There may be crosstalk with the estrogen receptor and human epidermal growth factor receptor-2 signal pathways in the metabolic regulation in breast cancer cells that make it more complex to evaluate the efficiency of an anti-metabolic drug. On the other hand, the research on target metabolism in breast cancer will also largely help us to understand the complicated mechanism by which an anti-metabolic drug improves the efficacy of cancer therapy or overcomes drug resistance.
Publish Date 2016-01-30 17:42
Citation Long JP, Li XN, Zhang F. Targeting metabolism in breast cancer: How far we can go? World J Clin Oncol 2016; 7(1): 122-130
Url http://www.wjgnet.com/2218-4333/full/v7/i1/122.htm
DOI http://dx.doi.org/10.5306/wjco.v7.i1.122
Full Article (PDF) WJCO-7-122.pdf
Full Article (Word) WJCO-7-122.doc
Manuscript File 20142-Review.docx
Answering Reviewers 20142-Answering reviewers.pdf
Audio Core Tip 20142-Audio core tip.mp3
Conflict-of-Interest Disclosure Form 20142-Conflict-of-interest statement.pdf
Copyright License Agreement 20142-Copyright assignment.pdf
Peer-review Report 20142-Peer-review(s).pdf
Scientific Misconduct Check 20142-Scientific misconduct check.pdf
Scientific Editor Work List 20142-Scientific editor work list.pdf