Publication Name	World Journal of Gastrointestinal Oncology
Manuscript ID	25816
Country	Japan

Received	2016-03-24 10:03
Peer-Review Started	2016-03-24 15:21
To Make the First Decision	2016-05-23 09:54
Return for Revision	2016-05-23 14:43
Revised	2016-06-08 14:48
Second Decision	2016-07-04 17:09
Accepted by Journal Editor-in-Chief
Accepted by Company Editor-in-Chief	2016-07-18 10:52
Articles in Press	2016-07-18 10:52
Publication Fee Transferred
Edit the Manuscript by Language Editor
Typeset the Manuscript	2016-08-27 17:30
Publish the Manuscript Online	2016-09-12 10:43

ISSN	1948-5204 (online)
Open Access	This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/
Copyright	© The Author(s) 2016. Published by Baishideng Publishing Group Inc. All rights reserved.
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Category	Gastroenterology & Hepatology
Manuscript Type	Review
Article Title	Pathogenesis and risk factors for gastric cancer after Helicobacter pylori eradication
Manuscript Source	Invited Manuscript
All Author List	Reina Ohba and Katsunori Iijima
Funding Agency and Grant Number
Corresponding Author	Reina Ohba, MD, PhD, Department of Gastroenterology, Akita University Graduate School of Medicine, 1-1-1 Hondo, Akita 010-8543, Japan. reina@doc.med.akita-u.ac.jp
Key Words	Helicobacter pylori; Eradication; Atrophic gastritis; Intestinal metaplasia; Metachronous gastric cancer
Core Tip	Helicobacter pylori (H. pylori) eradication and endoscopic resection appeared to reduce the risk of gastric cancer. However, recent studies show that the risk of metachronous gastric cancer increases in the background of gastric mucosal atrophy even after successful eradication. Thus, curing H. pylori infections may not prevent metachronous gastric cancer in background mucosa with intestinal metaplasia. We review the risk factors and possible approaches for reducing the risk of metachronous gastric cancer.
Publish Date	2016-09-12 10:43
Citation	Ohba R, Iijima K. Pathogenesis and risk factors for gastric cancer after Helicobacter pylori eradication. World J Gastrointest Oncol 2016; 8(9): 663-672
URL	http://www.wjgnet.com/1948-5204/full/v8/i9/663.htm
DOI	http://dx.doi.org/10.4251/wjgo.v8.i9.663

Full Article (PDF)	WJGO-8-663.pdf
Full Article (Word)	WJGO-8-663.doc
Manuscript File	25816-Review.docx
Answering Reviewers	25816-Answering reviewers.pdf
Audio Core Tip	25816-Audio core tip.mp3
Conflict-of-Interest Disclosure Form	25816-Conflict-of-interest statement.pdf
Copyright License Agreement	25816-Copyright assignment.pdf
Non-Native Speakers of English Editing Certificate	25816-Language certificate.pdf
Peer-review Report	25816-Peer-review(s).pdf
Scientific Misconduct Check	25816-Scientific misconduct check.pdf
Scientific Editor Work List	25816-Scientific editor work list.pdf