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11/10/2016 9:27:00 AM | Browse: 527 | Download: 370
Publication Name World Journal of Diabetes
Manuscript ID 25843
Country/Territory Spain
Received
2016-03-24 08:48
Peer-Review Started
2016-03-25 13:52
To Make the First Decision
2016-05-13 16:28
Return for Revision
2016-05-14 11:28
Revised
2016-07-16 18:48
Second Decision
2016-09-02 10:58
Accepted by Journal Editor-in-Chief
2016-09-02 14:03
Accepted by Company Editor-in-Chief
2016-09-08 16:18
Articles in Press
2016-09-08 16:18
Publication Fee Transferred
Edit the Manuscript by Language Editor
Typeset the Manuscript
2016-11-03 15:09
Publish the Manuscript Online
2016-11-10 09:27
ISSN 1948-9358 (online)
Open Access This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/
Copyright © The Author(s) 2016. Published by Baishideng Publishing Group Inc. All rights reserved.
Article Reprints For details, please visit: http://www.wjgnet.com/bpg/gerinfo/247
Permissions For details, please visit: http://www.wjgnet.com/bpg/gerinfo/207
Publisher Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA
Website http://www.wjgnet.com
Category Endocrinology and Metabolism
Manuscript Type Review
Article Title Nutrition, insulin resistance and dysfunctional adipose tissue determine the different components of metabolic syndrome
Manuscript Source Invited Manuscript
All Author List Juan Antonio Paniagua
Funding Agency and Grant Number
Corresponding author Juan Antonio Paniagua, PhD, Insulin Resistance, Metabolism and Adipose Tissue Unit, Maimonides Institute of Biomedical Research, University Hospital Reina Sofia, Avda Menendez Pidal, s/n, 14004 Cordoba, Spain. japaniaguag@yahoo.es
Keywords Obesity; Metabolic syndrome; Metabolism; Adipokines; Insulin resistance; Lipotoxicity and nutrition
Core Tip Central obesity, the insulin resistance, hyperglycemia, hypertriglyceridemia, hypoalphalipoproteinemia, hypertension and fatty liver are grouped in the so-called metabolic syndrome (MetS). In subjects with MetS an energy balance is critical to maintain a healthy body weight, mainly limiting the intake of high energy density foods. However, high-carbohydrate rich (CHO) diets increase postprandial peaks of insulin and glucose. Triglyceride-rich lipoproteins are also increased, which interferes with reverse cholesterol transport lowering high-density lipoprotein cholesterol. In addition, CHO-rich diets could move fat from peripheral to central deposits and reduce adiponectin activity in peripheral adipose tissue. All these are improved with monounsaturated fatty acid-rich diets. Lastly, increased portions of ω-3 and ω-6 fatty acids also decrease triglyceride levels, and complement the healthy diet that is recommended in patients with MetS.
Publish Date 2016-11-10 09:27
Citation Paniagua JA. Nutrition, insulin resistance and dysfunctional adipose tissue determine the different components of metabolic syndrome. World J Diabetes 2016; 7(19): 483-514
Url http://www.wjgnet.com/1948-9358/full/v7/i19/483.htm
DOI http://dx.doi.org/10.4239/wjd.v7.i19.483
Full Article (PDF) WJD-7-483.pdf
Full Article (Word) WJD-7-483.doc
Manuscript File 25843-Review.doc
Answering Reviewers 25843-Answering reviewers.pdf
Audio Core Tip 25843-Audio core tip.mp3
Conflict-of-Interest Disclosure Form 25843-Conflict-of-interest statement.pdf
Copyright License Agreement 25843-Copyright assignment.pdf
Supplementary Material Supplementary_Material_20160324030500.doc
Peer-review Report 25843-Peer-review(s).pdf
Journal Editor-in-Chief Review Report 25843-Journal editor-in-chief review report.pdf
Scientific Misconduct Check 25843-Scientific misconduct check.pdf
Scientific Editor Work List 25843-Scientific editor work list.pdf