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Articles Published Processes
7/29/2016 8:32:00 PM | Browse: 1295 | Download: 1777
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Received |
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2016-03-28 09:38 |
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Peer-Review Started |
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2016-03-28 15:30 |
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To Make the First Decision |
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2016-05-30 09:20 |
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Return for Revision |
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2016-05-30 13:00 |
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Revised |
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2016-06-12 07:30 |
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Second Decision |
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2016-06-23 17:26 |
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Accepted by Journal Editor-in-Chief |
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2016-06-24 07:06 |
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Accepted by Executive Editor-in-Chief |
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2016-07-06 18:38 |
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Articles in Press |
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2016-07-06 18:39 |
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Publication Fee Transferred |
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Edit the Manuscript by Language Editor |
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Typeset the Manuscript |
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2016-07-26 09:13 |
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Publish the Manuscript Online |
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2016-07-29 20:32 |
ISSN |
1007-9327 (print) and 2219-2840 (online) |
Open Access |
This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/ |
Copyright |
© The Author(s) 2016. Published by Baishideng Publishing Group Inc. All rights reserved.
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Article Reprints |
For details, please visit: http://www.wjgnet.com/bpg/gerinfo/247
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Permissions |
For details, please visit: http://www.wjgnet.com/bpg/gerinfo/207
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Publisher |
Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA |
Website |
http://www.wjgnet.com |
Category |
Gastroenterology & Hepatology |
Manuscript Type |
Topic Highlights |
Article Title |
Inflammatory microenvironment contributes to epithelial-mesenchymal transition in gastric cancer
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Manuscript Source |
Invited Manuscript |
All Author List |
Hui-Ying Ma, Xin-Zhou Liu and Chun-Min Liang |
Funding Agency and Grant Number |
Funding Agency |
Grant Number |
National Science Foundation of China |
3147114 |
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Corresponding Author |
Chun-Min Liang, MD, PhD, Lab of Tumor Immunology, Department of Anatomy and Histology and Embryology, Shanghai Medical College of Fudan University, 138 Yixueyuan Road, Shanghai 200032,
China. cmliang@fudan.edu.cn
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Key Words |
Gastric cancer; Inflammation; Epithelial-mesenchymal transition; Microenvironment; Immune cells |
Core Tip |
The major cause of gastric cancer (GC) is Helicobacter pylori infection, resulting in an inflam-matory microenvironment in GC. Meanwhile, the leading cause of death for GC patients is metastasis. The major pathway for metastasis is the epithelial-mesenchymal transition (EMT). Therefore, a thorough understanding of how the inflammatory microenvironment contributes to the promotion of the EMT is indispensable for develo-ping new treatments. In this review, we summarize the mechanisms of inflammatory mediators, divided among immune cells and molecules, on the prognosis of GC patients and EMT, which suggests that a combination of immunotherapy and anti-EMT treatments may be encouraging for the treatment of GC. |
Publish Date |
2016-07-29 20:32 |
Citation |
Ma HY, Liu XZ, Liang CM. Inflammatory microenvironment contributes to epithelial-mesenchymal transition in gastric cancer. World J Gastroenterol 2016; 22(29): 6619-6628 |
URL |
http://www.wjgnet.com/1007-9327/full/v22/i29/6619.htm |
DOI |
http://dx.doi.org/10.3748/wjg.v22.i29.6619 |
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