Publication Name	World Journal of Cardiology
Manuscript ID	38228
Country	South Africa

Received	2018-02-08 06:44
Peer-Review Started	2018-02-08 09:16
To Make the First Decision	2018-03-30 06:21
Return for Revision	2018-04-04 01:05
Revised	2018-04-06 12:23
Second Decision	2018-04-19 00:13
Accepted by Journal Editor-in-Chief
Accepted by Executive Editor-in-Chief	2018-04-22 17:40
Articles in Press	2018-04-22 17:40
Publication Fee Transferred
Edit the Manuscript by Language Editor
Typeset the Manuscript	2018-09-29 09:14
Publish the Manuscript Online	2018-09-30 06:52

ISSN	1949-8462 (online)
Open Access	This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/
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Category	Cardiac & Cardiovascular Systems
Manuscript Type	Minireviews
Article Title	Established and novel pathophysiological mechanisms of pericardial injury and constrictive pericarditis
Manuscript Source	Unsolicited Manuscript
All Author List	Vinasha Ramasamy, Bongani M Mayosi, Edward D Sturrock and Mpiko Ntsekhe
ORCID	Author(s) ORCID Number Vinasha Ramasamy http://orcid.org/0000-0002-3336-5773 Bongani M Mayosi http://orcid.org/0000-0001-6641-8950 Edward D Sturrock http://orcid.org/0000-0003-2032-7636 Mpiko Ntsekhe http://orcid.org/0000-0002-0851-7675
Funding Agency and Grant Number
Corresponding Author	Mpiko Ntsekhe, FACC, MD, MPhil, PhD, Professor, Division of Cardiology, University of Cape Town, Anzio Road, Observatory 7925, South Africa. mpiko.ntsekhe@uct.ac.za
Key Words	Inflammatory pericarditis; Autoimmune disease; Tuberculous pericarditis; Fibrosis mechanism; Constrictive pericarditis
Core Tip	Constrictive pericarditis arises as a complication of pericarditis from a wide range of aetiologies. A comprehensive understanding of the fibrotic process eventually leading to pathological symptoms is currently lacking.Through this review of the literature, we have identified various molecular mediators which are likely to play a role in the establishment of constriction and which warrant further studies.
Publish Date	2018-09-30 06:52
Citation	Ramasamy V, Mayosi BM, Sturrock ED, Ntsekhe M. Established and novel pathophysiological mechanisms of pericardial injury and constrictive pericarditis. World J Cardiol 2018; 10(9): 87-96
URL	http://www.wjgnet.com/1949-8462/full/v10/i9/87.htm
DOI	http://dx.doi.org/10.4330/wjc.v10.i9.87

Full Article (PDF)	WJC-10-87.pdf
Full Article (Word)	WJC-10-87.doc
Manuscript File	38228-Review.docx
Answering Reviewers	38228-Answering reviewers.pdf
Audio Core Tip	38228-Audio core tip.mp3
Conflict-of-Interest Disclosure Form	38228-Conflict-of-interest statement.pdf
Copyright License Agreement	38228-Copyright assignment.pdf
Approved Grant Application Form(s) or Funding Agency Copy of any Approval Document(s)	38228-Grant application form(s).pdf
Peer-review Report	38228-Peer-review(s).pdf
Scientific Editor Work List	38228-Scientific editor work list.pdf