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Articles Published Processes
9/10/2014 7:40:00 PM | Browse: 1139 | Download: 828
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Received |
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2013-06-27 14:14 |
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Peer-Review Started |
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2013-06-27 18:05 |
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To Make the First Decision |
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2013-07-26 18:31 |
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Return for Revision |
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2013-08-01 12:10 |
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Revised |
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2013-08-22 09:33 |
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Second Decision |
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2013-09-03 14:27 |
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Accepted by Journal Editor-in-Chief |
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Accepted by Executive Editor-in-Chief |
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2013-09-04 07:33 |
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Articles in Press |
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Publication Fee Transferred |
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Edit the Manuscript by Language Editor |
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Typeset the Manuscript |
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2013-10-08 16:28 |
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Publish the Manuscript Online |
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2013-10-11 13:29 |
Category |
Medicine, General & Internal |
Manuscript Type |
Review |
Article Title |
Adipose tissue dysfunction and the pathogenesis of metabolic syndrome
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Manuscript Source |
Invited Manuscript |
All Author List |
Di Zhao and Hui Liu |
Funding Agency and Grant Number |
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Corresponding Author |
Di Zhao, MD, PhD, Distinguished Professor of Zhengzhou University, Division of Hypertension, the Second Hospital Affiliated to Zhengzhou University, 2 Jingba Street, Zhengzhou 450014, Henan Province, China. emaildizhao@yahoo.com |
Key Words |
Metabolic syndrome; Adipose tissue dysfunction; Insulin resistance; Inflammation; Oxidative stress |
Core Tip |
Metabolic syndrome is a growing research area. Insulin resistance, obesity, inflammation and oxidative stress may play an important role in the pathogenesis of metabolic syndrome. The role of adipose tissue dysfunction is emphasized during the development of obesity in recent years. The study around adipose tissue dysfunction will help to understand the pathogenesis of metabolic syndrome and may bring effective therapy in treatment of metabolic syndrome related diseases. Therefore, this review mainly focuses on the roles of adipose tissue dysfunction in inflammation, insulin resistance, and oxidative stress in the pathogenesis of metabolic syndrome. |
Publish Date |
2013-10-11 13:29 |
Citation |
Zhao D, Liu H. Adipose tissue dysfunction and the pathogenesis of metabolic syndrome. World J Hypertens 2013; 3(3): 18-26 |
URL |
http://www.wjgnet.com/2220-3168/full/v3/i3/18.htm |
DOI |
http://dx.doi.org/10.5494/wjh.v3.i3.18 |
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