Publication Name	World Journal of Gastroenterology
Manuscript ID	7169
Country	South Korea

Received	2013-11-06 14:25
Peer-Review Started	2013-11-06 20:25
To Make the First Decision	2013-12-16 18:11
Return for Revision	2013-12-20 17:37
Revised	2014-01-15 16:17
Second Decision	2014-05-19 11:02
Accepted by Journal Editor-in-Chief
Accepted by Company Editor-in-Chief	2014-05-19 11:24
Articles in Press	2014-05-23 10:27
Publication Fee Transferred
Edit the Manuscript by Language Editor	2014-06-23 23:30
Typeset the Manuscript	2014-08-21 17:29
Publish the Manuscript Online	2014-09-07 13:31

ISSN	1007-9327 (print) and 2219-2840 (online)
Open Access
Copyright
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Publisher	Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA
Website	http://www.wjgnet.com

Category	Gastroenterology & Hepatology
Manuscript Type	Autobiography
Article Title	Osthol attenuates hepatic steatosis via decreased triglyceride synthesis not by insulin resistance
Manuscript Source	Invited Manuscript
All Author List	Ho Hyun Nam, Dae Won Jun, Hye Joon Jeon, Jai Sun Lee, Waqar Khalid Saeed and Eun Kyung Kim
Funding Agency and Grant Number	Funding Agency Grant Number National Research Foundation of Korea 2011-0007127
Corresponding Author	Dae Won Jun, MD, Department of Internal Medicine, Hanyang University School of Medicine, Hangdang dong 17,133-070, Seongdong-gu, Seoul 133-791, South Korea. noshin@hanyang.ac.kr
Key Words	Osthol; Non-alcoholic fatty liver disease; Sterol regulatory element binding protein
Core Tip	Nonalcoholic fatty liver disease is considered as a consequence of “multi-hit” processes. Osthol, a coumarin compound, has anti-inflammatory effects on various diseases. However, there is no multi-faceted and comprehensive evaluation of its effects. The current study evaluated effects of osthol on intrahepatic fat synthesis, ?-oxidation, inflammation, and insulin resistance by multifaceted analysis.
Publish Date	2014-09-07 13:31
Citation	Nam HH, Jun DW, Jeon HJ, Lee JS, Saeed WK, Kim EK. Osthol attenuates hepatic steatosis via decreased triglyceride synthesis not by insulin resistance. World J Gastroenterol 2014; 20(33): 11753-11761
URL	http://www.wjgnet.com/1007-9327/full/v20/i33/11753.htm
DOI	http://dx.doi.org/10.3748/wjg.v20.i33.11753

Full Article (PDF)	WJG-20-11753.pdf
Full Article (Word)	WJG-20-11753.doc
Manuscript File	7169-Review.docx
Answering Reviewers	7169-Answering review.pdf
Copyright License Agreement	7169-Copyright assignment.pdf
Peer-review Report	7169-Peer review.pdf
Scientific Editor Work List	7169-Scientific editor work list.pdf