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Articles Published Processes
9/1/2014 11:47:00 AM | Browse: 891 | Download: 841
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Received |
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2013-11-08 19:48 |
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Peer-Review Started |
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2013-11-09 20:44 |
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To Make the First Decision |
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2014-02-13 16:29 |
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Return for Revision |
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2014-02-20 09:08 |
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Revised |
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2014-02-28 14:03 |
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Second Decision |
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2014-03-18 14:58 |
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Accepted by Journal Editor-in-Chief |
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Accepted by Executive Editor-in-Chief |
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2014-03-18 15:19 |
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Articles in Press |
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Publication Fee Transferred |
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Edit the Manuscript by Language Editor |
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Typeset the Manuscript |
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2014-04-11 09:53 |
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Publish the Manuscript Online |
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2014-04-18 11:34 |
Category |
Cardiac & Cardiovascular Systems |
Manuscript Type |
Review |
Article Title |
Origin and therapy for hypertriglyceridaemia in type 2 diabetes
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Manuscript Source |
Invited Manuscript |
All Author List |
Jing Pang, Dick C Chan and Gerald F Watts |
Funding Agency and Grant Number |
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Corresponding Author |
Gerald F Watts, DSc, MD, PhD, FRACP, FRCP, School of Medicine and Pharmacology, University of Western Australia, GPO Box X2213, Perth, WA 6847, Australia. gerald.watts@uwa.edu.au |
Key Words |
Diabetes; Triglyceride; Therapy |
Core Tip |
Diabetic dyslipidemia relates collectively to hyperglycaemia, insulin resistance, hyperinsulinaemia, abdominal visceral adipose disposition, increased liver fat content, and dysregulated fatty acid metabolism. Insulin resistance in diabetes induces hypertriglyceridaemia by increasing the enterocytic production of chylomicrons and an impaired clearance capacity is also involved. Usual care for diabetic dyslipidemia is statin treatment, but a significant proportion of patients have residual dyslipidemia, related to hypertriglyceridaemia and atherogenic dyslipidemia. Current evidence supports the use of fenofibrate in type 2 diabetics with high triglyceride levels. |
Publish Date |
2014-04-18 11:34 |
Citation |
Pang J, Chan DC, Watts GF. Origin and therapy for hypertriglyceridaemia in type 2 diabetes. World J Diabetes 2014; 5(2): 165-175 |
URL |
http://www.wjgnet.com/1948-9358/full/v5/i2/165.htm |
DOI |
http://dx.doi.org/10.4239/wjd.v5.i2.165 |
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