Publication Name	World Journal of Gastroenterology
Manuscript ID	8357
Country	China

Received	2013-12-26 17:10
Peer-Review Started	2013-12-27 10:12
To Make the First Decision	2014-01-09 14:51
Return for Revision	2014-01-17 15:33
Revised	2014-02-09 20:22
Second Decision	2014-04-03 10:19
Accepted by Journal Editor-in-Chief
Accepted by Executive Editor-in-Chief	2014-04-03 12:37
Articles in Press	2014-05-23 09:22
Publication Fee Transferred
Edit the Manuscript by Language Editor	2014-04-18 19:26
Typeset the Manuscript	2014-06-17 17:59
Publish the Manuscript Online	2014-07-03 15:30

ISSN	1007-9327 (print) and 2219-2840 (online)
Open Access
Copyright
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Publisher	Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA
Website	http://www.wjgnet.com

Category	Gastroenterology & Hepatology
Manuscript Type	Case Control Study
Article Title	Interactions between pork consumption, CagA status and IL-1B-31 genotypes in gastric cancer
Manuscript Source	Unsolicited Manuscript
All Author List	Xiao-Qin Wang, Paul D Terry, Li Cheng, Hong Yan, Jian-Sheng Wang, Wen-An Wu and Sen-Ke Hu
Funding Agency and Grant Number	Funding Agency Grant Number Health Department of Shaanxi Province 2009K12-02
Corresponding Author	Xiao-Qin Wang, Associate Professor, Department of Statistics and Epidemiology, School of Medicine, Xi’an Jiaotong University, 76 West Yanta Road, Xi’an 710061, Shaanxi Province, China. wangxiaoqin@mail.xjtu.edu.cn
Key Words	Gastric cancer; Pork; CagA; interleukin-1B; Interaction; Helicobacter pylori
Core Tip	It is widely known that infectious, dietary, and genetic factors are implicated in gastric carcinogenesis, which is a long, complicated, and multi-stage process. The Helicobacter pylori (H. pylori) virulence factor CagA has been shown to be polymorphic and to contribute to disease pathogenesis in an allele-dependent manner. The interleukin (IL)-1 gene plays an important role in determining the long-term outcome of H. pylori infection. Dietary factors such as pork consumption may contribute to the malignancy process in synergy with these genetic factors and infectious agents. Our study further explores potential interactions among dietary (pork intake), infectious (H. pylori CagA positive) and genetic factors (IL-1B-31 genotypes) on gastric cancer risk.
Publish Date	2014-07-03 15:30
Citation	Wang XQ, Terry PD, Cheng L, Yan H, Wang JS, Wu WA, Hu SK. Interactions between pork consumption, CagA status and IL-1B-31 genotypes in gastric cancer. World J Gastroenterol 2014; 20(25): 8151-8157
URL	http://www.wjgnet.com/1007-9327/full/v20/i25/8151.htm
DOI	http://dx.doi.org/10.3748/wjg.v20.i25.8151

Full Article (PDF)	WJG-20-8151.pdf
Full Article (Word)	WJG-20-8151.doc
Manuscript File	8357-Review.doc
Answering Reviewers	8357-Answering reviewers.pdf
Copyright License Agreement	8357-Copyright assignment.pdf
Non-Native Speakers of English Editing Certificate	8357-Language certificate.pdf
Peer-review Report	8357-Peer reviews.pdf
Scientific Editor Work List	8357-Scientific editor work list.doc