Publication Name	World Journal of Diabetes
Manuscript ID	95192
Country	Brazil

Received	2024-04-03 13:31
Peer-Review Started	2024-04-07 00:29
To Make the First Decision
Return for Revision	2024-05-02 18:36
Revised	2024-05-11 20:33
Second Decision	2024-05-31 02:45
Accepted by Journal Editor-in-Chief
Accepted by Executive Editor-in-Chief	2024-05-31 06:50
Articles in Press	2024-05-31 06:50
Publication Fee Transferred
Edit the Manuscript by Language Editor	2024-06-13 17:53
Typeset the Manuscript	2024-07-28 13:24
Publish the Manuscript Online	2024-08-27 15:11

ISSN	1948-9358 (online)
Open Access	This article is an open-access article that was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution NonCommercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: https://creativecommons.org/Licenses/by-nc/4.0/
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Category	Endocrinology & Metabolism
Manuscript Type	Editorial
Article Title	Inflammatory markers, oxidative stress, and mitochondrial dynamics: Repercussions on coronary artery disease in diabetes
Manuscript Source	Invited Manuscript
All Author List	José Carlos Tatmatsu-Rocha and Luan Santos Mendes-Costa
ORCID	Author(s) ORCID Number José Carlos Tatmatsu-Rocha http://orcid.org/0000-0003-4900-4857 Luan Santos Mendes-Costa http://orcid.org/0000-0001-8767-7986
Funding Agency and Grant Number
Corresponding Author	José Carlos Tatmatsu-Rocha, MSc, PhD, Professor, Research Scientist, College of Medicine, ostgraduate Program in Physiotherapy and Functionality, Federal University of Ceara-UFC, 1127 Coronel Nunes de Melo, Fortaleza 60430-450, Ceará, Brazil. tatmatsu@ufc.br
Key Words	Mitochondrial dynamics; Diabetes; Oxidative stress; Coronary artery disease; Nε-carboxymethyl-lysine
Core Tip	Increased oxidative stress promoted by pro-inflammatory mediators such as Nε-carboxymethyl-lysine causes changes in mitochondrial dynamics and has been associated with insulin resistance and cardiovascular diseases. Therapies that promote the health of mitochondria by balancing the mechanisms of mitochondrial fusion and fission may be a path forward in the context of coronary artery disease.
Publish Date	2024-08-27 15:11
Citation	<p>Tatmatsu-Rocha JC, Mendes-Costa LS. Inflammatory markers, oxidative stress, and mitochondrial dynamics: Repercussions on coronary artery disease in diabetes. <i>World J Diabetes</i> 2024; 15(9): 1853-1857</p>
URL	https://www.wjgnet.com/1948-9358/full/v15/i9/1853.htm
DOI	https://dx.doi.org/10.4239/wjd.v15.i9.1853

Full Article (PDF)	WJD-15-1853-with-cover.pdf
Full Article (Word)	WJD-15-1853.docx
Manuscript File	95192-Review-Filipodia.docx
Answering Reviewers	95192-answering-reviewers.pdf
Audio Core Tip	95192-audio.m4a
Conflict-of-Interest Disclosure Form	95192-conflict-of-interest-statement.pdf
Copyright License Agreement	95192-copyright-assignment.pdf
Non-Native Speakers of English Editing Certificate	95192-non-native-speakers.pdf
Peer-review Report	95192-peer-reviews.pdf
Scientific Misconduct Check	95192-scientific-misconduct-check.png
Scientific Editor Work List	95192-scientific-editor-work-list.pdf
CrossCheck Report	95192-crosscheck-report.pdf