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Publication Name World Journal of Virology
Manuscript ID 22098
Country/Territory Colombia
2015-08-12 08:42
Peer-Review Started
2015-08-13 14:28
To Make the First Decision
2015-09-28 11:31
Return for Revision
2015-10-06 11:39
2015-10-23 01:52
Second Decision
2016-01-20 16:30
Accepted by Journal Editor-in-Chief
2016-01-22 14:36
Accepted by Company Editor-in-Chief
2016-01-29 17:14
Articles in Press
2016-01-29 17:15
Publication Fee Transferred
Edit the Manuscript by Language Editor
Typeset the Manuscript
2016-04-13 11:30
Publish the Manuscript Online
2016-05-06 13:48
ISSN 2220-3249 (online)
Open Access This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/
Copyright © The Author(s) 2016. Published by Baishideng Publishing Group Inc. All rights reserved.
Article Reprints For details, please visit: http://www.wjgnet.com/bpg/gerinfo/247
Permissions For details, please visit: http://www.wjgnet.com/bpg/gerinfo/207
Publisher Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA
Website http://www.wjgnet.com
Category Virology
Manuscript Type Review
Article Title Inflammatory and oxidative stress in rotavirus infection
Manuscript Source Invited Manuscript
All Author List Carlos A Guerrero and Orlando Acosta
Funding Agency and Grant Number
Corresponding Author Carlos A Guerrero, MD, MSc, PhD, Professor of Medicine, Department of Physiological Sciences, Faculty of Medicine, Universidad Nacional de Colombia, Carrera 45 # 26-85, Bogotá 111311, Colombia. caguerrerof@unal.edu.co
Key Words Rotaviruses; Oxidative stress; Inflammatory signaling; Antioxidant treatment; Anti-inflammatory treatment
Core Tip Rotavirus entry into the host cell requires cell surface molecules providing binding, chaperoning and oxido-reducing functions. Sialic acid/integrin a2b1, heat shock cognate protein 70 and protein disulfide isomerase (PDI) seem to perform these functions. Recently, the cell surface oxido-reduction activity based at least on PDI has been highlighted as a potential determinant of the conformational changes that are required by viral structural proteins in order to facilitate virus entry. The rotavirus-induced oxidative stress and inflammatory signaling is an attractive target for therapeutic intervention as antioxidant and anti-inflammatory treatment has proved to efficiently inhibit rotavirus infection.
Publish Date 2016-05-06 13:48
Citation Guerrero CA, Acosta O. Inflammatory and oxidative stress in rotavirus infection. World J Virol 2016; 5(2): 38-62
URL http://www.wjgnet.com/2220-3249/full/v5/i2/38.htm
DOI http://dx.doi.org/10.5501/wjv.v5.i2.38
Full Article (PDF) WJV-5-38.pdf
Full Article (Word) WJV-5-38.doc
Manuscript File 22098-Review.docx
Answering Reviewers 22098-Answering reviewers.pdf
Audio Core Tip 22098-Audio core tip.mp3
Conflict-of-Interest Disclosure Form 22098-Conflict-of-interest statement.pdf
Copyright License Agreement 22098-Copyright assignment.pdf
Non-Native Speakers of English Editing Certificate Editing_Certificate_20150812001950.docx
Peer-review Report 22098-Peer-review(s).pdf
Journal Editor-in-Chief Review Report 22098-Journal editor-in-chief review report.pdf
Scientific Misconduct Check 22098-Scientific misconduct check.pdf
Scientific Editor Work List 22098-Scientific editor work list.pdf