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9/12/2014 8:41:00 PM | Browse: 692 | Download: 833
Publication Name World Journal of Gastroenterology
Manuscript ID 4892
Country China
Received
2013-07-31 10:18
Peer-Review Started
2013-07-31 16:41
To Make the First Decision
2013-08-21 18:52
Return for Revision
2013-08-27 10:07
Revised
2013-09-05 12:16
Second Decision
2013-09-16 17:36
Accepted by Journal Editor-in-Chief
Accepted by Company Editor-in-Chief
2013-09-17 06:37
Articles in Press
Publication Fee Transferred
Edit the Manuscript by Language Editor
Typeset the Manuscript
2013-10-25 20:49
Publish the Manuscript Online
2013-11-16 18:29
ISSN 1007-9327 (print) and 2219-2840 (online)
Open Access
Copyright
Article Reprints For details, please visit: http://www.wjgnet.com/bpg/gerinfo/247
Permissions For details, please visit: http://www.wjgnet.com/bpg/gerinfo/207
Publisher Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA
Website http://www.wjgnet.com
Category Gastroenterology & Hepatology
Manuscript Type Review
Article Title Potential role of Helicobacter pylori infection in nonalcoholic fatty liver disease
Manuscript Source Invited Manuscript
All Author List Meng Li, Zhe Shen and You-Ming Li
Funding Agency and Grant Number
Funding Agency Grant Number
National Natural Science Foundation of China 81230012
Zhejiang Provincial Laboratory Animal Science Technology Program of China 2011C37088
National Natural Science Foundation of China 81300303
Corresponding Author You-Ming Li, Professor, Department of Gastroenterology, The First Affiliated Hospital, Zhejiang University School of Medicine, No. 79 Qingchun Road, Hangzhou 310003, Zhejiang Province, China. zliyouming@126.com
Key Words Helicobacter pylori; Nonalcoholic fatty liver disease; Insulin resistance; Inflammation; Cytokines
Core Tip A growing body of evidence suggests that Helicobacter pylori (H. pylori) infection is linked with nonalcoholic fatty liver disease (NAFLD). There are some potential pathogenic mediators and mechanisms involved in this progress, including fetuin-A, tumor necrosis factor-? and adiponectin. Long-term H. pylori infection may cause insulin resistance and inflammation, contributing to NAFLD. H. pylori toxins in the portal circulation arising from the gastroduodenal area may be another intriguing point, which might be related to the increased intestinal permeability in patients with NAFLD. It is hoped that eradication of H. pylori will provide a new treatment strategy for NAFLD.
Publish Date 2013-11-16 18:29
Citation Li M, Shen Z, Li YM. Potential role of Helicobacter pylori infection in nonalcoholic fatty liver disease. World J Gastroenterol 2013; 19(41): 7024-7031
URL http://www.wjgnet.com/1007-9327/full/v19/i41/7024.htm
DOI http://dx.doi.org/10.3748/wjg.v19.i41.7024
Full Article (PDF) WJG-19-7024.pdf
Manuscript File 4892-Review.doc
Answering Reviewers 4892-Answering Reviewers.pdf
Copyright License Agreement 4892-Copyright assignment.pdf
Non-Native Speakers of English Editing Certificate 4892-Language certificate.PDF
Peer-review Report 4892-Peer reviewer(s).pdf
Scientific Editor Work List 4892-Scientific editor work list.doc