Publication Name	World Journal of Biological Chemistry
Manuscript ID	18806
Country	Japan

Received	2015-04-28 18:26
Peer-Review Started	2015-04-29 16:13
To Make the First Decision	2015-05-14 16:45
Return for Revision	2015-05-19 09:40
Revised	2015-05-31 10:06
Second Decision	2015-06-24 08:29
Accepted by Journal Editor-in-Chief	2015-06-24 09:15
Accepted by Executive Editor-in-Chief	2015-07-02 16:29
Articles in Press	2015-07-02 16:30
Publication Fee Transferred
Edit the Manuscript by Language Editor
Typeset the Manuscript	2015-08-11 14:00
Publish the Manuscript Online	2015-08-26 11:07

ISSN	1949-8454 (online)
Open Access	This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/
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Category	Oncology
Manuscript Type	Review
Article Title	SGF29 and Sry pathway in hepatocarcinogenesis
Manuscript Source	Invited Manuscript
All Author List	Nobuya Kurabe, Shigekazu Murakami and Fumio Tashiro
Funding Agency and Grant Number	Funding Agency Grant Number “Academic Frontier” project for Private University 2006-2010 (to Tashiro F)
Corresponding Author	Dr. Fumio Tashiro, Department of Biological Science and Technology, Faculty of Industrial Science and Technology, Tokyo University of Science, Niijuku 6-3-1, Katsushika-ku, Tokyo 125-8585, Japan. ftashir@rs.noda.tus.ac.jp
Key Words	c-Myc; SPT3-TAF9-GCN5-acetyltransferase complex; SGF29; Sry; Hepatocarcinogenesis
Core Tip	Deregulated c-Myc expression is a hallmark of many human cancers. We have recently identified a role of mammalian homolog of yeast SPT-ADA-GCN5-acetyltransferas (SAGA) complex component, SAGA-associated factor 29 (SGF29), in regulating the c-Myc overexpression. We propose that the upstream regulation of SGF29 elicited by sex-determining region Y (Sry) is also augmented in hepatocellularcarcinoma (HCC). We hypothesize that c-Myc elevation driven by the deregulated Sry and SGF29 pathway is implicated in the male specific acquisition of human HCCs.
Publish Date	2015-08-26 11:07
Citation	Kurabe N, Murakami S, Tashiro F. SGF29 and Sry pathway in hepatocarcinogenesis. World J Biol Chem 2015; 6(3): 139-147
URL	http://www.wjgnet.com/1949-8454/full/v6/i3/139.htm
DOI	http://dx.doi.org/10.4331/wjbc.v6.i3.139

Full Article (PDF)	WJBC-6-139.pdf
Full Article (Word)	WJBC-6-139.doc
Manuscript File	18806-Review.docx
Answering Reviewers	18806-Answering reviewers.pdf
Audio Core Tip	18806-Audio core tip.mp3
Conflict-of-Interest Disclosure Form	18806-Conflict-of-interest statement.pdf
Copyright License Agreement	18806-Copyright assignment.pdf
Peer-review Report	18806-Peer-review(s).pdf
Journal Editor-in-Chief Review Report	18806-Journal editor-in-chief review report.pdf
Scientific Misconduct Check	18806-Scientific misconduct check.pdf
Scientific Editor Work List	18806-Scientific editor work list.pdf