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1/6/2016 8:46:00 PM | Browse: 578 | Download: 684
Publication Name World Journal of Nephrology
Manuscript ID 19484
Country/Territory Italy
2015-05-10 15:16
Peer-Review Started
2015-05-12 16:34
To Make the First Decision
2015-06-03 14:54
Return for Revision
2015-06-09 12:13
Second Decision
2015-11-30 09:36
Accepted by Journal Editor-in-Chief
2015-11-30 16:08
Accepted by Company Editor-in-Chief
2015-12-11 16:08
Articles in Press
2015-12-11 16:08
Publication Fee Transferred
Edit the Manuscript by Language Editor
Typeset the Manuscript
2015-12-28 15:47
Publish the Manuscript Online
2016-01-06 20:46
ISSN 2220-6124 (online)
Open Access This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/
Copyright © The Author(s) 2016. Published by Baishideng Publishing Group Inc. All rights reserved.
Article Reprints For details, please visit: http://www.wjgnet.com/bpg/gerinfo/247
Permissions For details, please visit: http://www.wjgnet.com/bpg/gerinfo/207
Publisher Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA
Website http://www.wjgnet.com
Category Urology and Nephrology
Manuscript Type Minireviews
Article Title Secondary amyloidosis in autoinflammatory diseases and the role of inflammation in renal damage
Manuscript Source Invited Manuscript
All Author List Roberto Scarpioni, Marco Ricardi and Vittorio Albertazzi
Funding Agency and Grant Number
Corresponding author Roberto Scarpioni, MD, Department of Nephrology and Dialysis, AUSL Hospital “Guglielmo da Saliceto”, via Taverna 49, 29121 Piacenza, Italy. rscarpioni@hotmail.com
Keywords Inflammation; Autoinflammatory disease; Chronic kidney disease; Interleukin-1; Dialysis; Caspase; Proteinuria; Amyloidosis; Nod-like receptor protein-3
Core Tip Inflammation may also negatively produce elevation of proinflammatory cytokines. Recently, attention was addressed to the formation of the intracellular inflammasome nod-like receptor protein-3 (NLRP-3) activating caspase-1, the enzyme required for the maturation of interleukin-1. IL1, in turn, regulate serum amyloid A, a major acute-phase with also proinflammatory properties. An interesting new scenario on the pathogenesis of renal diseases (namely ANCA-associated glomerulonephritis vasculitis, urate-crystal nephropathy, contrast nephropathy, acute kidney injury, reactive systemic amyloidosis) and reactive systemic amyloidosis was opened, and NLRP3 inflammasome was recently identified as a possible therapeutic target in the treatment of chronic kidney disease.
Publish Date 2016-01-06 20:46
Citation Scarpioni R, Ricardi M, Albertazzi V. Secondary amyloidosis in autoinflammatory diseases and the role of inflammation in renal damage. World J Nephrol 2016; 5(1): 66-75
Url http://www.wjgnet.com/2220-6124/full/v5/i1/66.htm
DOI http://dx.doi.org/10.5527/wjn.v5.i1.66
Full Article (PDF) WJN-5-66.pdf
Full Article (Word) WJN-5-66.doc
Manuscript File 19484-Review.docx
Answering Reviewers 19484-Answering reviewers.pdf
Audio Core Tip 19484-Audio core tip.m4a
Conflict-of-Interest Disclosure Form 19484-Conflict-of-interest statement.pdf
Copyright License Agreement 19484-Copyright assignment.pdf
Peer-review Report 19484-Peer-review(s).pdf
Journal Editor-in-Chief Review Report 19484-Journal editor-in-chief review report.pdf
Scientific Misconduct Check 19484-Scientific misconduct check.pdf
Scientific Editor Work List 19484-Scientific editor work list.pdf