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Articles Published Processes
9/1/2014 3:42:00 PM | Browse: 991 | Download: 1020
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Received |
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2013-01-10 08:59 |
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Peer-Review Started |
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2013-01-11 10:00 |
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To Make the First Decision |
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2013-03-04 11:50 |
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Return for Revision |
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2013-03-08 11:55 |
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Revised |
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Second Decision |
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2013-03-28 17:26 |
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Accepted by Journal Editor-in-Chief |
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Accepted by Executive Editor-in-Chief |
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2013-03-29 11:59 |
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Articles in Press |
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Publication Fee Transferred |
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Edit the Manuscript by Language Editor |
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Typeset the Manuscript |
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2013-04-12 10:06 |
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Publish the Manuscript Online |
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2013-04-15 20:21 |
Category |
Biochemistry & Molecular Biology |
Manuscript Type |
Minireviews |
Article Title |
Diabetes and renal tubular cell apoptosis
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Manuscript Source |
Invited Manuscript |
All Author List |
Samy L Habib |
Funding Agency and Grant Number |
Funding Agency |
Grant Number |
American Heart Association |
to Habib SL |
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Corresponding Author |
Samy L Habib, MS, PhD, Associate Professor, South Texas Veterans Health Care System, Departments of Cellular and Structural Biology, University of Texas Health Science Center, 7703 Floyd Curl Dr., San Antonio, TX 78229, United States. habib@uthscsa.edu |
Key Words |
Tubular cells; Renal; Apoptosis; Diabetes |
Core Tip |
Apoptosis contributes to the development of diabetic nephropathy, but the mechanism by which high glucose induces apoptosis is not fully understood. High glucose similar to those seen with hyperglycemia in people with diabetes mellitus, lead to accelerated apoptosis, a form of programmed cell death characterized by cell shrinkage, chromatin condensation and DNA fragmentation, in variety of cell types, including renal proximal tubular epithelial cells. |
Publish Date |
2013-04-15 20:21 |
Citation |
Habib SL. Diabetes and renal tubular cell apoptosis. World J Diabetes 2013; 4(2): 27-30 |
URL |
http://www.wjgnet.com/1948-9358/full/v4/i2/27.htm |
DOI |
http://dx.doi.org/10.4239/wjd.v4.i2.27 |
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